Hair Loss (MPB and Female Baldness) - Skull Expansion is the TRUE Cause! Part 2
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Genetic Hair Loss - Read This, Learn the Truth!
This lens reveals skull expansion as the TRUE cause of male pattern baldness (MPB) and hair thinning in women, and how the hair loss industry got it WRONG! Hair loss pictures show you exactly how the skull expansion process causes this distressing condition to develop for millions of men and women.
In Part 1, I explained how the underlying mechanism for androgenic alopecia remains unclear to the hair loss industry, and raised six very important questions about the hair loss process:
Q1. Why does remnant hair sometimes continue to grow within the MPB region despite extensive hair loss all around it?
Q2. What causes the same male pattern profile (horseshoe shape) to develop in almost all severe cases of androgenic alopecia?
Q3. How can DHT be linked to both hair loss and hair growth?
Q4. What is the genetic connection to androgenic alopecia?
Q5. Why does the rate of hair loss vary from person to person? (Up to 20% of men can suffer rapid hair loss starting as early as puberty. But most don't experience androgenic alopecia until later on in life in a much more gradual process.
Q6. Why does the location of hair loss vary within the MPB region? Some people only lose hair from the front (temple recession) or back of the scalp (a bald patch), whilst others lose hair from both these regions simultaneously?
Every one of these questions MUST be answered before any theory can be recognised as the true underlying mechanism for this type of hair loss. By reading this lens you'll find out exactly how skull expansion causes androgenic alopecia and provides answers to ALL these questions.
In Part 1, I explained how the underlying mechanism for androgenic alopecia remains unclear to the hair loss industry, and raised six very important questions about the hair loss process:
Q1. Why does remnant hair sometimes continue to grow within the MPB region despite extensive hair loss all around it?
Q2. What causes the same male pattern profile (horseshoe shape) to develop in almost all severe cases of androgenic alopecia?
Q3. How can DHT be linked to both hair loss and hair growth?
Q4. What is the genetic connection to androgenic alopecia?
Q5. Why does the rate of hair loss vary from person to person? (Up to 20% of men can suffer rapid hair loss starting as early as puberty. But most don't experience androgenic alopecia until later on in life in a much more gradual process.
Q6. Why does the location of hair loss vary within the MPB region? Some people only lose hair from the front (temple recession) or back of the scalp (a bald patch), whilst others lose hair from both these regions simultaneously?
Every one of these questions MUST be answered before any theory can be recognised as the true underlying mechanism for this type of hair loss. By reading this lens you'll find out exactly how skull expansion causes androgenic alopecia and provides answers to ALL these questions.
New Table of Contents
Genetic Hair Loss
Androgenic Alopecia
Androgenic alopecia - also known as androgenetic alopecia, AGA, male pattern baldness (MPB) and female pattern baldness. Whatever you call it (I used to call it "the bane of my life") androgenic alopecia is a condition, not a disease - even though it might feel like one!
As you can see from these hair loss pictures, this type of hair loss affects both men and women (in fact, up to 80% of men and 50% of women will experience severe hair loss at some point in their lives!)
These photos can now help explain how skull expansion causes androgenic alopecia:
As you can see from these hair loss pictures, this type of hair loss affects both men and women (in fact, up to 80% of men and 50% of women will experience severe hair loss at some point in their lives!)
These photos can now help explain how skull expansion causes androgenic alopecia:
Skull Expansion
Bone Resorption and Remodelling
Skull expansion is related to the bone resorption and remodelling processes.
When you're growing up, your skeleton obviously gets bigger and bigger. This, of course, includes the skull, which not only grows in size, but also changes shape.
Once you reach adulthood, bone resorption and remodelling will continue to maintain skeletal integrity. Together, these two essential processes constantly renew the entire skeleton throughout life. For some, this simply means maintaining the bones - keeping them strong and healthy. But, for those who suffer androgenic alopecia, certain bones of the skull will continue to grow.
This is skull expansion, and is the direct cause of androgenic alopecia within the MPB region of the scalp for both men and women. It can also be considered as an exaggeration of the bone resorption and remodelling processes.
Scalp Capillary Network
For all those with the genetic predisposition towards skull expansion/ androgenic alopecia, this process will progressively stretch and pull tight the scalp tissue that overlies the skull. This constricts the blood vessels, and with it, reduces their blood supply.
Figure 3 shows how the main arterial network of the scalp will provide a strong blood supply to the back and sides. But, within the MPB region, the follicles are only served by a much weaker capillary network (not shown in the diagram). This illustrates how the MPB region is the most likely area of the scalp to suffer a reduction in blood supply.
Within the MPB region, this reduction in blood flow will decrease the supply of nutrients required by follicles to grow hair. And, over time, hair follicle miniaturisation and hair loss will result.
Development of the Male Pattern Profile
If you compare Figure 3 with Figure 4, you can see that the bones of the skull that underlie the MPB region are the frontal and parietal bones.
Figure 5 details these two bones. The frontal and parietal eminences represent the areas where ossification commenced (in other words, where bone initially began growing as a foetus within the womb). From these areas, bone grows outwards, pushing androgenic alopecia inst the surrounding bone tissue and so causing the frontal and parietal bones to grow larger in surface area. It's through this linear growth that the adult skull size is eventually reached.
There are two parietal bones (left and right) so each one obviously has its own parietal eminence.
But, you can also see that there are two frontal eminences as well. That's because the frontal bone actually starts out as two bones within the womb. As these develop, they gradually fuse together and can leave a residual suture line* (as shown in Figure 5). This will usually disappear as a baby grows into a child, but the two frontal eminences remain.
* Suture lines mark the connection between the bones of the skull.
Skull expansion of these two frontal eminences (left and right) will cause hair loss at the front hairline. This explains why, in most cases, hair recedes at the left and right temples and not simultaneously across the entire front hairline.
It also explains why some remnant hair often continues to grow at the front - you can see this in Figure 2 and Figure 6. (Basically, remnant hair will continue to grow wherever there is no, or little, skull expansion).
This provides the answer to question 1.
In Figure 6 you can see how the frontal and parietal bones of the skull relate to the pattern of hair loss for someone with severe androgenic alopecia.
Now, can you see how the temporal ridge seems to coincide with hair loss along the sides of the head?
Well, this is no coincidence!
Within each parietal bone, the parietal eminence (that causes skull expansion) only lies above the temporal ridge. So, it logically follows that hair loss can also only occur above this line. And, as a result, the temporal ridge marks an approximate boundary between hair loss and hair growth on the sides of the head.
Hair will always continue to grow at the lower back of the head because here, the occipital bone largely remains unaffected by parietal bone expansion. You can see the occipital bone in Figure 4).
By analysing of the structure of the skull (specifically the frontal eminences, temporal ridge and occipital bone), the familiar male pattern profile of hair loss can be explained.
This provides the answer to question 2.
When you're growing up, your skeleton obviously gets bigger and bigger. This, of course, includes the skull, which not only grows in size, but also changes shape.
Once you reach adulthood, bone resorption and remodelling will continue to maintain skeletal integrity. Together, these two essential processes constantly renew the entire skeleton throughout life. For some, this simply means maintaining the bones - keeping them strong and healthy. But, for those who suffer androgenic alopecia, certain bones of the skull will continue to grow.
This is skull expansion, and is the direct cause of androgenic alopecia within the MPB region of the scalp for both men and women. It can also be considered as an exaggeration of the bone resorption and remodelling processes.
Scalp Capillary Network
For all those with the genetic predisposition towards skull expansion/ androgenic alopecia, this process will progressively stretch and pull tight the scalp tissue that overlies the skull. This constricts the blood vessels, and with it, reduces their blood supply.
Figure 3 shows how the main arterial network of the scalp will provide a strong blood supply to the back and sides. But, within the MPB region, the follicles are only served by a much weaker capillary network (not shown in the diagram). This illustrates how the MPB region is the most likely area of the scalp to suffer a reduction in blood supply.
Within the MPB region, this reduction in blood flow will decrease the supply of nutrients required by follicles to grow hair. And, over time, hair follicle miniaturisation and hair loss will result.
Development of the Male Pattern Profile
If you compare Figure 3 with Figure 4, you can see that the bones of the skull that underlie the MPB region are the frontal and parietal bones.
Figure 5 details these two bones. The frontal and parietal eminences represent the areas where ossification commenced (in other words, where bone initially began growing as a foetus within the womb). From these areas, bone grows outwards, pushing androgenic alopecia inst the surrounding bone tissue and so causing the frontal and parietal bones to grow larger in surface area. It's through this linear growth that the adult skull size is eventually reached.
There are two parietal bones (left and right) so each one obviously has its own parietal eminence.
But, you can also see that there are two frontal eminences as well. That's because the frontal bone actually starts out as two bones within the womb. As these develop, they gradually fuse together and can leave a residual suture line* (as shown in Figure 5). This will usually disappear as a baby grows into a child, but the two frontal eminences remain.
* Suture lines mark the connection between the bones of the skull.
Skull expansion of these two frontal eminences (left and right) will cause hair loss at the front hairline. This explains why, in most cases, hair recedes at the left and right temples and not simultaneously across the entire front hairline.
It also explains why some remnant hair often continues to grow at the front - you can see this in Figure 2 and Figure 6. (Basically, remnant hair will continue to grow wherever there is no, or little, skull expansion).
This provides the answer to question 1.
In Figure 6 you can see how the frontal and parietal bones of the skull relate to the pattern of hair loss for someone with severe androgenic alopecia.
Now, can you see how the temporal ridge seems to coincide with hair loss along the sides of the head?
Well, this is no coincidence!
Within each parietal bone, the parietal eminence (that causes skull expansion) only lies above the temporal ridge. So, it logically follows that hair loss can also only occur above this line. And, as a result, the temporal ridge marks an approximate boundary between hair loss and hair growth on the sides of the head.
Hair will always continue to grow at the lower back of the head because here, the occipital bone largely remains unaffected by parietal bone expansion. You can see the occipital bone in Figure 4).
By analysing of the structure of the skull (specifically the frontal eminences, temporal ridge and occipital bone), the familiar male pattern profile of hair loss can be explained.
This provides the answer to question 2.
Hormones and Hair Loss
Androgens, like DHT, can be linked to both hair loss and hair growth. DHT causes facial, body and pubic hair to grow in men during puberty, but it also has a direct connection with androgenic alopecia.
How can this be?
DHT is a steroid hormone which means that, whilst it stimulates new hair growth, it also has an anabolic effect on bone formation. In other words, it makes bone grow! And it's through this continued bone growth (skull expansion) that overwhelms the hair growth promoting effects of DHT. This provides the answer to question 3.
Steroid hormones are responsible for the rapid increase in bone growth and muscle development in men at puberty. This also explains why bodybuilders often suffer androgenic alopecia - intense weight training will increase testosterone and DHT levels (as do anabolic steroids).
Women generally experience androgenic alopecia to a much lesser extent than men due to their much higher oestrogen and lower testosterone levels.
Whereas steroid hormones like testosterone and its derivatives have an anabolic effect on bone growth, oestrogen decreases the bone resorption process. Since the oestrogen levels in a healthy pre-menopausal woman's body greatly exceed those of testosterone, there will be a reduced tendency towards skull expansion.
However, low (post-menopausal) oestrogen levels can often lead to androgenic alopecia (see Figure 1). That's because testosterone levels will be higher (relative to the lower oestrogen levels) and so the bone resorption, remodelling and skull expansion processes will all increase for those women with the genetic predisposition towards androgenic alopecia.
So, for all those men and women with this genetic predisposition, DHT will cause the frontal and parietal eminences to grow during adulthood. This is the true underlying mechanism behind androgenic alopecia.
But why do some have the genetic predisposition towards skull expansion/ androgenic alopecia whilst others do not?
How can this be?
DHT is a steroid hormone which means that, whilst it stimulates new hair growth, it also has an anabolic effect on bone formation. In other words, it makes bone grow! And it's through this continued bone growth (skull expansion) that overwhelms the hair growth promoting effects of DHT. This provides the answer to question 3.
Steroid hormones are responsible for the rapid increase in bone growth and muscle development in men at puberty. This also explains why bodybuilders often suffer androgenic alopecia - intense weight training will increase testosterone and DHT levels (as do anabolic steroids).
Women generally experience androgenic alopecia to a much lesser extent than men due to their much higher oestrogen and lower testosterone levels.
Whereas steroid hormones like testosterone and its derivatives have an anabolic effect on bone growth, oestrogen decreases the bone resorption process. Since the oestrogen levels in a healthy pre-menopausal woman's body greatly exceed those of testosterone, there will be a reduced tendency towards skull expansion.
However, low (post-menopausal) oestrogen levels can often lead to androgenic alopecia (see Figure 1). That's because testosterone levels will be higher (relative to the lower oestrogen levels) and so the bone resorption, remodelling and skull expansion processes will all increase for those women with the genetic predisposition towards androgenic alopecia.
So, for all those men and women with this genetic predisposition, DHT will cause the frontal and parietal eminences to grow during adulthood. This is the true underlying mechanism behind androgenic alopecia.
But why do some have the genetic predisposition towards skull expansion/ androgenic alopecia whilst others do not?
Genetic Link to Androgenic Alopecia
It's long been known that a genetic link to androgenic alopecia exists.
Most research currently places focus on the 5-alpha reductase and androgen receptor genes. However, it's the genes that determine skull shape and size that are responsible for androgenic alopecia .
Sexual Dimorphism
Sexual dimorphism means that the form (shape, size, etc) can vary between the two sexes. For example, women usually have wider hips, whilst men are generally taller and heavier (due to bigger bones and greater muscle development).
Skull expansion is a largely sexually dimorphic characteristic, affecting men much more so than women. Men will, quite simply, grow a bigger skull than most women. (This reflects the higher androgen levels men have, as well as differences in genetic inheritance). More specifically to androgenic alopecia, this means that, for most women, the frontal and parietal bones will be proportionately smaller than in the majority of men.
Growth Potential
Associated with skull shape and size is its growth potential. This simply means that some skull shapes are more likely to grow than others.
For example, someone whose skull shape has a high growth potential will be especially prone to skull expansion, and so invariably develop severe androgenic alopecia. And it follows that, anyone else with a very similar skull shape and growth potential will most likely develop androgenic alopecia to the same extent.
Of course, very similar skull shapes often run in families, and you may already be aware that, if your mother or father lost their hair, you too have a very high chance of losing yours as well. (This explains the strong genetic connection that androgenic alopecia can have within a family, especially between fathers and their sons).
You now know that the genetically determined characteristics of skull shape and size form the genetic link to androgenic alopecia. This provides the answer to question 4.
Most research currently places focus on the 5-alpha reductase and androgen receptor genes. However, it's the genes that determine skull shape and size that are responsible for androgenic alopecia .
Sexual Dimorphism
Sexual dimorphism means that the form (shape, size, etc) can vary between the two sexes. For example, women usually have wider hips, whilst men are generally taller and heavier (due to bigger bones and greater muscle development).
Skull expansion is a largely sexually dimorphic characteristic, affecting men much more so than women. Men will, quite simply, grow a bigger skull than most women. (This reflects the higher androgen levels men have, as well as differences in genetic inheritance). More specifically to androgenic alopecia, this means that, for most women, the frontal and parietal bones will be proportionately smaller than in the majority of men.
Growth Potential
Associated with skull shape and size is its growth potential. This simply means that some skull shapes are more likely to grow than others.
For example, someone whose skull shape has a high growth potential will be especially prone to skull expansion, and so invariably develop severe androgenic alopecia. And it follows that, anyone else with a very similar skull shape and growth potential will most likely develop androgenic alopecia to the same extent.
Of course, very similar skull shapes often run in families, and you may already be aware that, if your mother or father lost their hair, you too have a very high chance of losing yours as well. (This explains the strong genetic connection that androgenic alopecia can have within a family, especially between fathers and their sons).
You now know that the genetically determined characteristics of skull shape and size form the genetic link to androgenic alopecia. This provides the answer to question 4.
Rate and Location of Hair Loss
Skull shape and its growth potential can account for the different rates of hair loss, and the location (within the MPB region) in which it occurs.
Rate
The rate at which you lose hair is directly related to the extent of your skull expansion. This, quite obviously means that, the more your skull expands, the more hair you're likely to lose.
For the 20% of men whose skull shape has a high growth potential, this explains why androgenic alopecia will rapidly start developing from puberty and can lead to extensive hair loss by the age of thirty. However, in most cases, androgenic alopecia won't start until later on in life and it will be a much more gradual process - these skull shapes have a low growth potential.
Growth potential can, therefore, account for the variations that exist in the rate of hair loss. This answers question 5.
Location
Within the MPB region, the location in which androgenic alopecia develops can vary.
For some, hair loss develops at the front (temple recession) or back of the scalp (a bald patch). For others, androgenic alopecia will affect both these areas simultaneously and will cause either diffuse thinning throughout the MPB region, or more concentrated hair loss (a bald patch at the back together with receding temples at the front).
Skull expansion of the frontal bone will form a receding hairline from the temples. Skull expansion of the parietal bones will cause hair loss at the back of the scalp. And skull expansion of the frontal and parietal bones simultaneously will create hair loss in the front and back of the scalp at the same time.
This answers question 6.
Rate
The rate at which you lose hair is directly related to the extent of your skull expansion. This, quite obviously means that, the more your skull expands, the more hair you're likely to lose.
For the 20% of men whose skull shape has a high growth potential, this explains why androgenic alopecia will rapidly start developing from puberty and can lead to extensive hair loss by the age of thirty. However, in most cases, androgenic alopecia won't start until later on in life and it will be a much more gradual process - these skull shapes have a low growth potential.
Growth potential can, therefore, account for the variations that exist in the rate of hair loss. This answers question 5.
Location
Within the MPB region, the location in which androgenic alopecia develops can vary.
For some, hair loss develops at the front (temple recession) or back of the scalp (a bald patch). For others, androgenic alopecia will affect both these areas simultaneously and will cause either diffuse thinning throughout the MPB region, or more concentrated hair loss (a bald patch at the back together with receding temples at the front).
Skull expansion of the frontal bone will form a receding hairline from the temples. Skull expansion of the parietal bones will cause hair loss at the back of the scalp. And skull expansion of the frontal and parietal bones simultaneously will create hair loss in the front and back of the scalp at the same time.
This answers question 6.
Convinced?
You should be!
Skull expansion is the true underlying mechanism behind androgenic alopecia. It has explained how, for those with the genetically determined characteristics of skull shape and size, DHT will cause the frontal and parietal eminences to grow during adulthood. It has also explained how DHT can be associated with both hair loss and hair growth, and provided answers to all six questions (something the current theory cannot do).
Now you know that skull expansion is the true cause of androgenic alopecia, the next question you need to ask is:
What skull shapes will lead to skull expansion?
Different skull shapes exist, and these determine where within the MPB region skull expansion and androgenic alopecia will occur, and how the rate of hair loss will vary.
But, to find out more about skull shape variations, you'll need to read the FREE ebook: "Skull Expansion - True Cause of Genetic Hair Loss". This ebook contains more information, more hair loss pictures and references that verify everything you've just read! You can get it here -
http://www.top-hair-loss-remedy.com/squidoo-2-hlr
How to stop thinning hair and hair loss?
Most natural remedies for thinning hair and alopecia simply DON'T work. That's because they don't treat the underlying cause of this condition - skull expansion.
You can read my story in this lens -
The Top Hair Loss Remedy for MPB and Female Baldness.
Thanks for reading,
Click here for Part 1 - Skull Expansion: True Cause of MPB and Female Baldness.
PS: If you're in any doubt about the skull expansion process causing androgenic alopecia, why not simply contact a hair specialist, ask those six questions, and see what answers you get!
Copyright © 2008 by Paul Taylor. All Rights Reserved.
Skull expansion is the true underlying mechanism behind androgenic alopecia. It has explained how, for those with the genetically determined characteristics of skull shape and size, DHT will cause the frontal and parietal eminences to grow during adulthood. It has also explained how DHT can be associated with both hair loss and hair growth, and provided answers to all six questions (something the current theory cannot do).
Now you know that skull expansion is the true cause of androgenic alopecia, the next question you need to ask is:
What skull shapes will lead to skull expansion?
Different skull shapes exist, and these determine where within the MPB region skull expansion and androgenic alopecia will occur, and how the rate of hair loss will vary.
But, to find out more about skull shape variations, you'll need to read the FREE ebook: "Skull Expansion - True Cause of Genetic Hair Loss". This ebook contains more information, more hair loss pictures and references that verify everything you've just read! You can get it here -
http://www.top-hair-loss-remedy.com/squidoo-2-hlr
How to stop thinning hair and hair loss?
Most natural remedies for thinning hair and alopecia simply DON'T work. That's because they don't treat the underlying cause of this condition - skull expansion.
You can read my story in this lens -
The Top Hair Loss Remedy for MPB and Female Baldness.
Thanks for reading,
Click here for Part 1 - Skull Expansion: True Cause of MPB and Female Baldness.
PS: If you're in any doubt about the skull expansion process causing androgenic alopecia, why not simply contact a hair specialist, ask those six questions, and see what answers you get!
Copyright © 2008 by Paul Taylor. All Rights Reserved.
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- get-your-hair-back get-your-hair-back May 31, 2009 @ 1:13 am
- Hair loss can be stopped through many natural ways.
* Herbal medicines are there to end your problems. If herbal medication does not work for you, talk to your doctor.
* Massage your hair. When you massage your hair, the blood flow on your head is increased. When the blood flow increases, your hair naturally regrows. Studies show that bald men have only 2% blood circulation on average, while men who grow their hair healthily have 10% blood circulation in their scalp.
* Nutritious foods prevent hair loss too, especially those that are rich in protein and have low carbohydrate content. While you can include fatty acids in your diet, like soy, fish, canola oil and walnut, you need to stay away from foods with high fat content. Omega 3 fatty acids are highly recommended since these have anti-inflammatory properties that play a major role in keeping your hair healthy.
Women Hair Loss Treatment
by pjtaylor
Hi,
I'm Paul Taylor - welcome to my profile.
I suffered genetic hair loss (male pattern baldness or androgenetic alopecia) for seven years before fi... (more)
I'm Paul Taylor - welcome to my profile.
I suffered genetic hair loss (male pattern baldness or androgenetic alopecia) for seven years before fi... (more)
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